Chapter
3
EVOLUTION
OF CULTURE
The
task is… not so much to see what no one has yet seen; but to think
what
nobody
has yet thought, about that which everybody sees.
—Erwin
Schrodinger
I
have been studying society my whole life and have noticed that people
are like a flock of birds that can change direction in flight all at
once in a perfectly orchestrated fashion by some hidden signaling system
they have developed. As a child my neighborhood revolved around a humble
little community where all the neighbors knew each other on a first-name
basis, but fifty years of misdirected cultural evolution has turned my
charming neighborhood into the hood. How did people change so much in
one generation? Culture is the soul of society. It has a mind of its own
to believe what it wants, yet the mind of culture is not subject to any
one person, because it doesn’t belong to anyone, but to the group,
which has the power to shape peoples’ behavior, making them act as one
person. Culture creates itself on a subliminal level, thinking and
acting collectively with no one seemingly at the wheel. Culture is so
close to the human experience that we would not be human without it.
This strange inherent phenomenon of group behavior, sown into the human
gene pool has proven itself central to humanity, always evolving and
having free reign to express itself in each successive generation, but
how do we control the ebb and flow of change when there doesn’t appear
to be anyone at the wheel? When society loses control of what they are
becoming and elects fate as their leader of cultural change, the least
common denominator always takes control and misdirects people into
shallow waters, invariably running them aground on the shoals of selfish
hedonism.
Chapter
one, Inventors of Culture, concluded that man is destined to destroy
himself through his technological advancements, which made life more
feasible, causing populations to soar, and making civilization a
requirement. Chapter two The Apostasy ended on the note that, despite
this technological age, the world is more lost than ever, because the
church has lost its own way. This chapter will suggest a mechanism that
powers human evolution, equating civilization with domestication,
suggesting that technology has made us soft, which has inadvertently
compromised the human gene pool, making it easier to lead the world to
its logical conclusion. This chapter will attempt to identify the
cultural communication device that drives human evolution by examining
how worldviews seep into our minds, are converted to biochemical
molecules that are genetically transferred to our offspring and reemerge
in our children as predispositions for certain behaviors, perpetuating
the cycle of cultural evolution.
It
is obvious that culture influences society, but it is more meaningful to
think in terms of how culture influences the individual. Accordingly, my
definition of “Culture” is: The collective influence of individuals
on society. By this definition we see that culture manages the mindset
of society, though it does not control the individual, since it
represents the group as a whole.
Back
in the 1950s a man named Alan Turing, an embryologist, designed a
computer program that could play checkers. He calculated that there were
1040 possible moves, so he realized he needed to write a program that
could select game winning moves among the vast number of possibilities,
but teaching a computer to play checkers is tricky business. He tried a
variety of approaches, and settled on a strategy that after tinkering
with it a few years eventually beat some of the best checker players of
all time. He came to the conclusion that he needed to design a program
that could write itself, supplying it with the game’s objective and a
set of rules to follow and then integrated a number of feedback loops
into it, so the program could evaluate its own progress and learn from
its mistakes. The more the program played, the more it tweaked its
strategy and improved its game. In his book Darwin’s Dangerous Idea
Daniel Dennett used this story as an analogy that non-living mater
discovered life on its own and then taught itself how to survive and
flourish (Dennett, pp. 208-212). In stark contrast to Dennett’s idea,
the checkers program needed a god to create it (Alan Turing) and then to
make it understand that it was playing a game of checkers in the first
place, but Dennett didn’t use Mr. Turing in his analogy, because
Dennett is an atheist. The checkers program especially needed Mr. Turing
to give it the rules of composing a strategy, so it could establish its
own algorithm from millions of possible tactics. When you include Mr.
Turing’s input, the story works very well as an analogy for creation,
but without Mr. Turing the story doesn’t make any sense. However, one
thing does make sense about Dennett’s analogy, God designed life in
very much the way that Mr. Turing designed his checkers program: He
designed it to take care of itself.
We
have seen animals obeying their instincts from childhood, such as when
fall comes and geese fly south for the winter. These animals are obeying
certain genetic switches that are linked to seasonal changes in sunlight
and temperature. In what manner did God give instincts to all the living
creatures? If He did it on the day of creation, then why do their
instincts change with the changing environment and appear to be tailor
made for their specific needs? God certainly created the heavens and the
earth, man and all the animals, but ever since the day of creation, the
earth and its inhabitants have never ceased to change. Adaptation then
as we observe it in nature should be viewed as developing shapes and
sizes, behaviors and instincts that are flexible enough to accommodate a
rapidly changing environment. The result is that it makes animals very
flexible (plastic) in their ability to adapt, because they can draw from
changes in their surroundings, or they can draw from their own genetic
past, or they can press forward in completely novel directions.
There
is compelling evidence that instincts and idiosyncratic behavior develop
through a mechanism that universally exists in the genetic fabric of all
living things, including man, with the purpose of acclimating to their
environment. This inborn mechanism that has several names but none that
is unanimously accepted by the scientific consensus belongs to a fast
growing field called Epigenetics. It best explains the marvelously
flexible system of checks and balances we observe in all living things,
picking its target of selection based on what the animal most often uses
for survival; for us it is our brains.
Before
the industrial revolution, the father passed down his trade to his sons.
Shoeman, Smith, Wheeler and Taylor are still common names that described
what people did for a living. Back then the son inherited the name along
with the trade, and after so many generations he also inherited an
aptitude and predisposition to carry on the family business. Teasing out
the differences between nature and nurture is not easy, but there are
examples in nature that totally eliminate the nurture side of this
equation. For example, how do salmon instinctively swim upstream in a
specific creek to lay their eggs in the same general location where they
were hatched without any instruction from their parents? A map of their
destination must have been hardwired into their little fish brains while
they were still in the egg. Does this happen only to salmon? How do some
migratory birds instinctively know which direction to fly and how do
they know when they have arrived at their destination, and how do they
even know they are migratory birds if they have never received
instruction from their parents? Again, perhaps everything they need to
know is formed in them while they were still in the egg? Do only certain
animals have these gifts, or are they the animals that make it crystal
clear that these things are happening in us all?
“…Genes
are followers, not leaders, in evolution” is a quote by Mary Jane
West-Eberhard in her book Developmental Plasticity and Evolution (p.
20), which is the theme of her work in genetics. The fact that genes do
not lead but follow suggests a very flexible genome and requires very
far-reaching and revolutionary changes in our thinking about genetics
that has been teaching us for the last century and a half that we are
the product of our genes. With a brain as big and powerful as our own it
would seem impossible that we could use it to point ourselves in the
wrong direction, but if all humanity throughout the ages could make one
set of footprints, you could look back and see the progress of a man who
appears to be totally lost. Society is disintegrating before our very
eyes and no one knows why or how to stop it.
Following
are three factors in order of influence that cycle through the
generations, making us who and what we are:
1)
Genetic traits we receive at birth
2)
Behaviors that form during our critical period from environmental
influences, between the ages of 0-5
3)
Influences of society (culture)
Understanding
how novel traits emerge in plants and animals would wrap up nicely if
there were any evidence to support the idea that our experiences
directly edit the DNA molecule, but they don’t. According to the
central dogma of genetics called Weismann’s Barrier, genetic
information passes in only one direction: from DNA → to RNA
→ to Protein. In other words protein molecules cannot “upload”
their information to DNA from which they originated. However, it is
hardly necessary for information to pass directly to the next generation
by editing germ cells, since there are other ways for this to happen
without risking the integrity of genetic material.
Geneticists
have calculated that about 98 percent of our genes do not code for
proteins, making them available for other kinds of work, such as cell
maintenance, much of which is epigenetic. Epigenetics – the prefix:
“Epi-” means that something is occurring at, near or against the
gene (American Heritage Dictionary) to the somatic cells of our bodies.
We have two kinds of cells: gamete (sex) cells, which we use to
reproduce that cannot be edited, and somatic (bone and tissue) cells
that are the composition of our bodies that are epigenetically modified
throughout our lives. In other words according to this definition,
something is occurring near, at or against the genes of somatic cells to
influence the way they express themselves. That is, DNA may code for
physical and behavioral traits, but Epigenetic molecules appended to the
DNA has the final say about what those physical and behavioral traits
will be! For example, when our brains develop during our critical period
between the ages of 0-5, it forms around environmental conditions of the
outside world. These environmental changes to our DNA are expressed by
epigenetic influences. The key question is: do we inherit these
epigenetic effects? If we do, then we can inherit environmental
influences on the gene. Eva Jablonka and Marion Lamb say we do.
…For
many years it was taken for granted that all memories of the
“epigenetic past” had to be completely erased before cells became
germ [reproductive] cells. This assumption ruled out any possibility
that induced epigenetic variations could be inherited. The discovery in
the 1980s that the epigenetic slate is not wiped clean—that some
epigenetic information does pass from one generation to the next—was
therefore totally unexpected (Jablonka, Page 139).
Epigenetic
functions do such things as assign flags to certain genes. These flags
(also termed switches) consist of small molecules that surround the
gene, creating a microenvironment around them, appending additional
instructions to their protein synthesis or else turning them on or off.
For example, kidney cells are kidney cells because epigenetic effects on
the cell turn on the sections of the chromosome that code for kidney
cell production (Jablonka, page 113). When that cell divides, the
resulting child cell is another kidney cell, which means epigenetic
effects are inherited from cell to cell, and as previously stated, there
is compelling evidence that they are inherited from parent to offspring:
In
genetic inheritance, traits are passed from one generation to the next
via DNA sequences in genes. Differences in a DNA sequence specify
differences in a trait. Epigenetic inheritance involves passing a trait
from one generation to the next without a difference in DNA sequence.
Known mechanisms of epigenetic inheritance include changes in molecular
structures around the DNA so that while the gene is the same, the gene
behaves differently. For example, genes switch on and off in response to
hormonal signals. [i]
There
is only a 1.1% difference in our DNA from chimpanzees, suggesting that
the major difference between them and us is epigenetic (West-Eberhard, p
335)! The way our genes function and express themselves are performed
under a giant web of epigenetic influences which do far more than anyone
expected in order to create the expressions of life and variation that
we observe in nature and in ourselves. One proof of this is the Human
Genome Project itself. Scientists were disappointed about half way
through the mission when they began to realize that what they were
learning was never going to span the immeasurable variation of
nature’s intricate complexity. The great hope of the Human Genome
Project was to plot one gene to one trait, and with that they could heal
any disease, modify our food production and genetically perfect
humanity, but there wasn’t a one-to-one correlation between genes and
proteins, and scientists quickly realized that DNA doesn’t fit into
the nice formula that they expected. There are only about ten thousand
genes in the human genome. That may sound like a lot, until you begin
adding up all the different tissues and organs throughout the body,
along with our physiological systems and anatomic structures, etc.
When
you consider that only about 2-3 percent of our genes are coding for
proteins, it reduces the number of active protein coding genes to about
300. The only possible explanation for the complexity of life under such
a small number of genes is that they are working in concert with each
other in a near infinite array of combinations. When you consider that
the possible combinations for 300 genes is about 3 X 10 384 (the number
3 with 384 zeros behind it), our observations of the rich variation of
life are restored. This means we must revise our earlier notions of one
gene coding for one protein. Genes mostly work in groups, are assigned
to groups at birth, and are reassigned and rearranged throughout life,
based on epigenetic information that attempts to fulfill the survival
needs of the organism, based on a built-in highly flexible genome that
God designed to respond directly to the environment. To the degree that
our genes are hardly different from that of a chimp is the degree to
which something vastly unintuitive is transpiring within our cells.
For
example, genetic information that no longer serves a purpose, usually
from a change in the environment, can be put aside as modules, and then
later un-shelved and reused at a later time (called reversion). These
modules are bundles of genes that combine forces capable of engineering
very specific physical and behavioral characteristics. Based on these
insights, biological processes interpret the way we live as adapting to
the environment, and then begin to modify the environment of our DNA to
accommodate highly tuned instincts to help us deal with a changing
world. We humans use the word culture in place of instincts.
In
development, genes and environment have complementary quantitative
effects on switches, such that an increase in the influence of one
implies a commensurate decrease in the influence of the other. There is
a sliding continuum in their proportional influences on switch
determination, such that environmental and genetic factors in a sense
compete with each other for control of regulation (West-Eberhard, p.
100).
In
other words, as we mature throughout childhood there is one of two
developmental pathways we will follow, depending on the similarities or
differences of the environment from that of our parents and their
ancestors. If the offspring is born and raised in an environment that is
similar to the parent’s experiences, they will develop closely forming
to the information contained in their DNA that they were given at birth,
but if the environment differs significantly, their DNA will search for
new developmental pathways that will attempt to acclimate to their
unique circumstances.
Homology
(Same-ology) is the study of biological similarities within all living
matter. For example, fish eyes work similar to bird eyes that work
similar to human eyes. Since there are so many similarities between
biological systems, it should not surprise anyone to know that there are
functional similarities between our DNA and the neurons of our brain.
Brain cells wire to other brain cells that fire in concert with the same
outside stimuli, referred to Hebbian Plasticity that holds to the
truism: ‘Cells that fire together wire together.’ Just as the brain
is incredibly flexible (especially in children) and continually rewires
itself throughout life to accommodate our mental and physiological
needs, so our genes are flexible in very similar ways. Our genes often
need to trade positions along the DNA strand, controlled by epigenetic
functions that reassign genes to work with other genes, causing them to
produce completely different expressions to accommodate our needs based
on our changing environment. In genetics jargon this process of gene
sequencing is called Exon Shuffling.
An
Exon as defined by the American Heritage Dictionary as: “A nucleotide
sequence in DNA that carries the code for the final messenger RNA
molecule and thus defines the amino acid sequence during protein
synthesis.” According to this definition, exons constitute the end
product of gene expression, so that when these units trade places the
body can make any protein it needs, with the goal of establishing highly
specific behavioral modules for the purpose of accommodation. Barlow
(1977) wrote: “A cornerstone of ethological theory is the belief that
behavior comes in discrete packets…” (referring to modularity p.
98), and as West-Eberhard also wrote, “Traits can be lost and regained
as units” (p. 234). So then, we think and behave in a manner similar
to our biology, but that shouldn’t surprise anyone. The process of
exon shuffling is partly summarized by West-Eberhard, p.317:
Since
biochemistry and molecular biology focus on the fundamentally modular
structure and behavior of biological molecules, it is perhaps not
surprising that they arrived early at a combinatorial view of evolution,
and that it was a molecular biologist (Jacob, 1977) who described
evolution as “tinkering” with preexisting pieces. The lowest level
of combinatorial evolution is based on the “changeability” of the
genetic code—its ability to undergo rearrangement without loss of
functionality (Maeshiro and Kimura, 1998).
Combinatorial
adaptation refers to groups of genes that form modules to produce new
protein molecules that alter physical and behavioral traits, effectively
fine-tuning the organism. Phenotypic plasticity is a popular scientific
term that refers to the flexibility of gene expression. In other words,
Genes
can produce alternate effects by differences in transcription from
different promoters. Another way is by differences in how the
transcripts are joined together, or spliced. RNA splicing cuts and then
joins together (splices) sections of transcribed pre-RNA into the mRNA
that will form the functional gene product. Alternative splicing
produces alternative molecular phenotypes, and as might be predicted, it
is involved in the origins of molecular novelties (West-Eberhard, pp.
318-323).
Now
that we understand that epigenetic functions are commonplace and
important, let’s take a closer look at what is happening around the
DNA molecule to make it respond so concisely to environmental conditions
when these switches are applied. Switches refer to, “Some element of a
phenotype [that] changes from a default state, action, or pathway to an
alternative one” (West-Eberhard, Page 67), referred to as an
Alternative Phenotype (or alternative gene expression). The greatest
strength of epigenetic switches is their ability to determine which
genes work in concert with other genes. Genes are assigned partners at
birth, but throughout our lives they trade partners with other genes in
an attempt to accommodate environmental demands, as mentioned earlier,
similar to dendrite reconfiguration within the brain as we mentally
adapt to our environment. Since there are so many similarities between
functional processes of DNA and mental processes within the brain,
perhaps they are indeed interconnected. Just as the eye is the window to
the soul, so maybe the brain is the window to our DNA. Perhaps our DNA
has the ability to observe the changes that take place within the brain
and use that information as a blueprint of its own genetic
rearrangement. Perhaps the regulating forces of epigenetic change are
looking to the brain as a model to tweak the somatic DNA molecule before
it reproduces itself in the next cell generation to reflect the changes
in the way we live. These alternative expressions are best seen as
genetic modules that are pre-built and self-contained components
designed for easy assembly and flexible use that can be invoked by a
single switch. “A switch implies some change in state, for example,
between on and off, under certain conditions… so a condition
sensitivity is an implicit quality of all switches. They mark
developmental decision points that depend on conditions. Conditions in
this case may refer to the internal environment, the social environment,
or the external environment” (West-Eberhard, p. 68).
There
is a well-known story about a Russian man named Dmitri Belyaev, who bred
silver foxes for tameness in Novosibirsk. Prior to his breeding efforts,
these foxes were afraid of people and would snarl at anyone who came too
close, or would hurt themselves trying to get away. Some would overheat
or suffocate from fear. Although they had been raising these foxes for
eighty years, they still had not become tame. People who have attempted
to raise wolves encountered exactly the same problems. Belyaev reported
that 40 percent of them were aggressively fearful, 30 percent were
extremely aggressive, 20 percent were fearful and 10 percent
“displayed a quiet, exploratory reaction without either fear or
aggression.” Belyaev separated the ten percent based on their flight
distance, which was measured by two attributes: 1) how close he could
get to the foxes before they ran away, and 2) how far away they would
run. It only took eighteen generations to tame these foxes, after which
they would look for their keepers and jump on their laps to get petted;
they would let people rub their bellies, let them give them their shots
and answered to their names. In short, they began to look and behave
like dogs. At the beginning of Mr. Belyaev’s selective breeding
experiment there was not a single fox that came close to being as tame
as the foxes that resulted from his efforts. As these foxes became tamer
other totally unexpected physical attributes began to emerge outside of
Mr. Belyaev’s selective control. In other words they were byproducts
of selection.
Their
tails turned up at the end, like a dog’s. Their coats were often
piebald [black and white spotted or patched], their ears drooped
[floppy], and the females came into heat twice a year instead of once.
Belyaev noted, “They even sound like dogs.” (Coppinger, P. 64)
…
but for the most part, while the dog is in its first few weeks of life,
and growing its brain, it is making the cell connections and rearranging
them in a specific way, according to the signals that are coming from
outside. This development predetermines its adult behavior. In other
words, imprinting changes the dog forever. (Coppinger, P. 105)
Understanding
brain growth should dispel the nature/nurture controversy once and for
all. It is never, ever either nature or nurture, but always both at the
same time. But liver cells make more liver cells because that is the
environment they respond to. Behavior is always epigenetic—above the
genes—an interaction between the genes and the developmental
environment. (Coppinger, P. 113)
It
is determined there is a definite look and behavior of tameness in the canine family, so is
there a definite look and behavior of tameness in humans— Tameness
being tantamount to Civilization? The short answer is yes. What are the
byproducts of domestication for us? Dogs go into heat twice a year
instead of once and have smaller skulls than wolves, suggesting that
being more sexually active and the loss of intelligence are natural
byproducts of tameness, because any creature that is tamed no longer
relies on its wits to survive, and if food is abundant, proliferating
will naturally increase. There are probably many other negative
byproducts of domestication, but the loss of intelligence I should think
would trump them all.
Remember
that Mendelian genetics cannot account for novel traits, including
tameness in these foxes. Pure Mendelian genetics modifies traits that
already exist and you simply get more of the same, but Epigenetics is
the road that leads to increased predispositions and novel traits. This
is why Mr. Belyaev not only selected for tameness in the foxes
(Mendelian Genetics), but also worked with them from pups during their
critical period to instill the characteristic tameness in them
(Epigenetics), just like you would create a guard dog or a bird dog from
a pup. In the process of raising these foxes from pups Mr. Belyaev
encouraged the epigenetic connections in their brains to form around the
characteristic tameness, and then selected the tamest foxes from the
litter for mating. In this way he combined genetics with epigenetics to
increase the measure of tameness in the foxes from one generation to the
next. This well documented account makes it crystal clear that some of
the epigenetic information that the foxes acquired during their critical
period was passed to the next generation and accumulated in the foxes’
gene pool through the course of selection. We humans also have a
critical period, between the ages of 0-5, and we also have a selection
regiment called culture. This critical period is when we develop
epigenetic connections within the brain that starts the process of
passing adaptive information to our DNA as the first step in
transcribing those traits to our offspring. Following is a summary of
the epigenetic process of inheritance:
Epigenetic Inheritance Cycle
-
Parents
epigenetically transcribe information from their own critical period
to the DNA of their children during conception, establishing
predispositions for behavior
-
Cognitive
development occurs within children between the ages of 0-5 (critical
period) based on family environment, supplementing or else
overriding the parents’ congenital input
-
Social
pressures from culture (gene pool) influence behavior throughout
adulthood, influencing how parents raise their children
-
Cultural
instincts form between the generations, entrenching consistent
traits into the DNA
*
*
*
*
*
*
Now
that we have established that there is a genetic mechanism encoding
predispositions into our DNA, we will now turn our attention back to the
subject we left behind at the end of chapter two, The Apostasy, and
apply what we learned from this chapter to the problems we see in the
church and in society as a whole. Going back to the old covenant
Levitical priesthood that consisted of the tribe of Levy, these priests
were selected by Israel based on their genealogy (only the tribe of Levy
could act as priests). This became a breeding ground for the epigenetic
inheritance cycle proposed above. However, the Catholic priesthood was
not handed down from father to son like the Levitical Priesthood, but
anybody could be a Catholic priest. In other words the present day
Catholic priesthood does not represent a single tribe, but a
cross-section of our contemporary society. It represents a cross-section
of the degenerating human gene pool, which is controlled by this
epigenetic inheritance cycle, that is, by culture. The nation of Israel
became so evil that Jesus had to come down as their messiah to fix their
problems and they crucified Him for it. In the last days the religions
of the world will become so atrocious that He will return again to fix
the problems, and they will attempt to kill Him again, only this time
God will judge the world for rejecting His offer of eternal life. Now,
listen to what Jesus said about the religious monsters of His day, and
compare His words to the monsters of our own time and to society as a
whole.
Woe
to you, teachers of the law and Pharisees, you hypocrites! You build
tombs for the prophets and decorate the graves of the righteous. And you
say, `If we had lived in the days of our forefathers, we would not have
taken part with them in shedding the blood of the prophets.' So you
testify against yourselves that you are the descendants of those who
murdered the prophets. Fill up, then, the measure of the sin of your
forefathers! "You snakes! You brood of vipers! How will you escape
being condemned to hell? Therefore I am sending you prophets and wise
men and teachers. Some of them you will kill and crucify; others you
will flog in your synagogues and pursue from town to town. And so upon
you will come all the righteous blood that has been shed on earth, from
the blood of righteous Abel to the blood of Zechariah son of Berekiah,
whom You murdered between the temple and the altar. I tell you the
truth, all this will come upon this generation. Mat 23,29-36 NIV
We
know according to Rom 5:12-21 that every person has genetically
inherited Adam’s sin. Therefore, it is no stretch to believe that
after reading the above verses we can inherit the sins of our
forefathers. Jesus accused the Pharisees of being sons of those who
murdered the prophets, but the ancient prophets were ancient even to the
Pharisees. Israel hadn’t seen a prophet in four hundred years (there
was a long prophetic silence before Christ), so there was no way the
Pharisees learned by example the murderous behavior of their fathers,
suggesting that the Pharisees’ great, great grandfathers who murdered
the prophets had an indirect influence on them. How can people act like
their ancestors whom they have never met?
Rejecting
the truth and killing Christ came naturally to the Pharisees through
genetically instilled predispositions, similar to instincts, according
to 2 Peter 2:12, “But these, like unreasoning animals, born as
creatures of instinct to be captured and killed, reviling where they
have no knowledge, will in the destruction of those creatures also be
destroyed,” NASB. When Jesus posed the rhetorical question to the
Pharisees, “How will you escape the sentence of hell?” He was basing
their judgment on their own sin, not on the sins of their forefathers.
Jesus was painting the picture of the Pharisees’ ancestors pushing
down their sins to their offspring, while the Pharisees were
simultaneously tugging their hatred of Jesus from their own chromosomes!
Jesus
spoke as though they were inherently guilty of being sons of their
fathers, as though being born into their respective families made them
guilty of murdering the Son of God, saying, “Fill up, then, the
measure of the sin of your forefathers!” There must be some truth to
this, otherwise Jesus would not have said it. He was referring in part
to ancestral sin. It doesn’t predestine anyone to act a certain way,
but it does predispose them to certain behaviors. Born and raised to
mirror their genetic code instinctively without thinking by reflex as
though trusting a voice that reverberates through their flesh as the
antithesis of the Holy Spirit. They feel they have a destiny to explore
their corrupt desires, born as reprobates and raised to fulfill their
predisposed demise with a propensity to follow the currents of their
fleshly desires, they move impetuously on command, having learned
obedience to carnal impulses from childhood.
Job
is feisty in his suffering and poses a question to God, “ [It is
said,] `God stores up a man's punishment for his sons.' Let him repay
the man himself, so that he will know it! (Job 21:19).” What Job
didn’t know was that the Pharisees were by no means innocent. So, it
seems that God judges the sons for emulating their fathers. The
Pharisees confidently boasted they were better than their forefathers
because they understood the prophets to be martyrs for telling the
truth, yet in their own hearts they believed that the only good prophet
was a dead one. It is one thing for the Pharisees to speak well of the
prophets, but quite another to stand in the presence of Jesus Christ,
who epitomized the Spirit of the prophets and murder Him for telling
them the truth, just like their fathers treated the prophets before
them. The ancient prophets were eventually exonerated, but while they
were alive, they “were stoned; they were sawed in two; they were put
to death by the sword. They went about in sheepskins and goatskins,
destitute, persecuted and mistreated- the world was not worthy of them.
They wandered in deserts and in mountains and in caves and holes in the
ground” (Heb 11:37-38 NIV).
In
review, Jesus sentenced the Pharisees to hell based on their genealogy,
which at least partially determined their behavior. Over a period of
many generations satanic powers work on cultures and subcultures,
weakening their will to resist sin, making them more and more evil and
slowly editing their hereditary makeup. Their polluted genes are then
passed from generation to generation ultimately accumulating in the
human gene pool with the goal of preparing humanity to receive their
antichrist in the last days. As an example, Jesus accused the Pharisees
of murdering Zechariah, who lived hundreds of years before the Pharisees
were even born. The Pharisees did not murder Zechariah, but Jesus held
them personally responsible for his blood, saying, “So upon you will
come all the righteous blood that has been shed on earth, from the blood
of righteous Abel to the blood of Zechariah son of Berekiah, whom you
murdered between the temple and the altar” (Mat 23:35). Jesus was
speaking both to the Pharisees and to the spirit of murder within them,
whom the Pharisees worshipped and served, who murdered the prophets and
all the righteous men from the beginning of time until now.
A
more accurate interpretation of this verse might be that He held their
generation personally responsible for all the blood spilled on the earth
since the beginning of time, saying, “I tell you the truth, all this
will come upon this generation” Mat 23:36. When Jesus was speaking to
the spirit of murder, He was prophesying about his own crucifixion. God
intended to heap upon Him the sin of the whole world committed since the
beginning of time, so that He might destroy the power of sin and break
the chains of ancestral regression perpetuated by demons for those who
would believe in Him through faith and receive eternal life. In other
words He offered the world a new beginning, and it worked for a while,
but now man has slumped back into a deep spiritual apathy where religion
is common as sandstone, but faith is rare as gold.
Sin
accumulates in the human gene pool, which is why God had to start over
many times throughout history. Had God not intervened during these
times, man would have continued in his wickedness until he destroyed
himself before the time. Following is a list of historical beginnings:
1. Noah’s ark
2. The tower of Babel
3. Jesus personally interceded for mankind by dying for our sins, giving
the world an opportunity to launch themselves into a new, positive
direction
4. The end times – it is not really the end of time, but the beginning of
a new age
Jesus
has already died and paid the price for man’s sin, but man has
continued in his legacy of depravity as though God never bestowed His
mercy upon us. It says He died once for all (Heb 7:27), so that, “If
we go on sinning willfully after receiving the knowledge of the truth,
there no longer remains a sacrifice for sins, but a terrifying
expectation of judgment, and the fury of a fire which will consume the
adversaries,” Heb 10:26-27 NASB. Let’s go back to Job’s statement
(Job 21:19); “God stores away a man’s iniquity for his sons.” The
way Jesus spoke about sin from Able to Zechariah, He made it sound like
it accumulates, inviting us to ask the question: what if all the sin of
man from which he has not repented has been accumulating in the human
gene pool since the crucifixion of Christ to the present day? When God
judges the sin of the world in the last days, He intends to make mankind
pick up the tab, since Jesus picked it up last time. Man has responded
to the gospel throughout the centuries by “trampling under foot the
Son of God, and have regarded as unclean the blood of the covenant by
which [they were] sanctified, and have insulted the Spirit of grace”
(Heb 10:29). “For if we go on sinning willfully after receiving the
knowledge of the truth, there no longer remains a sacrifice for sins”
(Heb 10:26). What else is God supposed to do for us after we have
rejected the offer of His Son? When you think of all the horrors
prophesied for the last days, collectively they will amount to the
horror that Jesus experienced on the cross. The fact that man has
collectively rejected the work of the cross is the very cause of God’s
impending judgment that He is about to unleash against the unbelieving
world in the last days.
James
R. Wuthrich
jimwuthrich@yahoo.com

Sources
1
http://www.ourstolenfuture.org/NewScience/reproduction/sperm/2005/2005-0602anwayetal.htm#epigenetic
2
http://km.typepad.com/index/story/
3
http://www.answers.com/topic/mendelian-inheritance
4
Jose Sanchez. Quoted in the New York Times, July 7,1989.
American
Heritage© Dictionary of the English Language, Third Edition
Copyright© 1992 by Houghton Mifflin Company. Electronic version
licensed from and portions copyright© 1994 by InfoSoft
International, Inc. All rights reserved.
Barlow,
G.W. (1977). Modal action patterns. In: How animals communicate,
T.A. Sebeok (ed.). Indiana University Press, Bloomington, pp. 98-134
Coppinger,
Raymond and Lorna. (2001). Dogs, A New Understanding of Canine
Origin, Behavior, and Evolution. University of Chicago Press.
Dennett,
Daniel. (1995). Darwin’s Dangerous Idea. Simon & Schuster
Paperbacks, New York.
Downes
, Stephen M. (2003) Evolution and Learning: The Baldwin Effect
Reconsidered. MIT Press. Cambridge, Massachusetts.
Fabian,
B. 1985. Ontogenetic explorations into the nature of evolutionary
change. In: Species and Speciation, E.S. Vrba (ed.). Transvaal
Museum Monograph No. 4. Transvaal Museum, Pretoria, pp. 77-85
Hare,
Robert D. (1993). Without Conscience: The disturbing world of the
psychopaths among us. The Guilford Press, New York (London,
England).
Hare
R. D. (1970). Psychopathy: Theory and Research. New York: Wiley.
Jablonka,
Eva and Lamb, Marion (2005). Evolution In Four Dimensions, genetic,
epigenetic, behavioral, and symbolic variation in the history of
life. MIT Press, Massachusetts (London, England).
Jacob,
F. 1977. Evolution and Tinkering. Science 196:1161-1166
Jackson,
R.R. and Wilcox, R.S. 1993. Spider flexibly chooses aggressive
mimicry signals for different prey by trial and error. Behavior
127:21-36
Kandel,
Eric R. (2006) In Search of Memory: The Emergence of a New Science
of Mind. W.W. Norton & Company, Inc. New York, NY.
Maeshiro,
T. and Kimura, M. 1998. The role of robustness and changeability on
the origin and evolution of genetic codes. Proceedings of the
National Academy of Sciences USA 95:5088-5093
Merriam-Webster's
Medical Dictionary. Merriam-Webster, Inc. http://dictionary.reference.com/browse/epigenetics
(accessed: January 09, 2007).
Parmesan,
C., Singer, M.C. and Harris, I. 1995. Absence of adaptive learning
from the oviposition foraging behavior of a checkerspot butterfly.
Animal Behavior 50:161-175
Rutter,
Michael. (2006) Genes and Behavior: Nature-Nurture Interplay
Explained. Blackwell Publishing. Oxford, UK.
Schwartz,
Jeffrey M. and Sharon Begley (2002). The Mind and the Brain:
Neuroplasticity and the Power of Mental Force. Harper-Perennial. New
York.
Stephens,
D.W. and Krebs, J.R. 1986. Foraging Theory. Princeton
University Press, Princeton
West-Eberhard,
Mary Jane (2003). Developmental Plasticity and Evolution. Oxford
University Press. New York, NY.